Fig_9.tif (1.08 MB)
Proliferation of primary activated HSCs was measured by crystal violet assay.
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posted on 2015-08-25, 03:07 authored by Ying-Hsien Huang, Mao-Meng Tiao, Li-Tung Huang, Jiin-Haur Chuang, Kuang-Che Kuo, Ya-Ling Yang, Feng-Sheng WangA miR-29a mimic or HDAC4 RNAi inhibited the proliferation of primary HSCs. In addition, treatment with a miR-29a anti-sense inhibitor significantly increased HSC proliferation. Data are expressed as the mean ± SE of four independent experiments. *indicates a p < 0.05 between the groups.
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HDAC 4Histone Deacetylases 4 BackgroundRecent studiesHDAC 4 functionprofibrogenic gene expressionhistone deacetylases 4migration assays.ResultsAfter BDLHSC migrationcholestatic liver fibrosisglial fibrillary acidic protein expressionprofibrogenic phenotypeantisense inhibitormirHSC activationHepatic Stellate CellsHSC proliferation.ConclusionsOur resultsActivated Hepatic Stellate Cells Modulates
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