pone.0210656.g004.tif (3.02 MB)
Changes in RAS enzyme activity in kidneys of mice with AAI-induced acute nephropathy.
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posted on 2019-01-11, 18:36 authored by Wen-Yeh Hsieh, Teng-Hsiang Chang, Hui-Fang Chang, Wan-Hsuan Chuang, Li-Che Lu, Chung-Wei Yang, Chih-Sheng Lin, Chia-Chu ChangRenal ACE (A), chymase (B) and ACE2 (C) activities were determined, and the related activity levels are shown as levels relative to the Control group. All values are expressed as the means ± SD from each group (n = 9). * and ** indicate p < 0.05 and p < 0.01 compared to the Control group, respectively; †† indicates p < 0.01 compared to the AAI group.
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ACEIangiotensin II formationp-Smad 3 levelsBWAng II levelAAI-induced nephropathy progressionAng II levelskidney injury developmentchymase hydrates AngRenal chymase-dependent pathwayACEaristolochic acid I-inducedCIAng IInephropathy Angiotensin-converting enzymeC 57BL miceAAI-treated miceroleAAI-induced nephropathic miceRenal RAS-related enzyme activities
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