10.1371/journal.pone.0210656.g006
Wen-Yeh Hsieh
Wen-Yeh
Hsieh
Teng-Hsiang Chang
Teng-Hsiang
Chang
Hui-Fang Chang
Hui-Fang
Chang
Wan-Hsuan Chuang
Wan-Hsuan
Chuang
Li-Che Lu
Li-Che
Lu
Chung-Wei Yang
Chung-Wei
Yang
Chih-Sheng Lin
Chih-Sheng
Lin
Chia-Chu Chang
Chia-Chu
Chang
The level of inflammatory signaling pathways in the kidneys of mice with AAI-induced acute nephropathy.
Public Library of Science
2019
ACEI
angiotensin II formation
p-Smad 3 levels
BW
Ang II level
AAI-induced nephropathy progression
Ang II levels
kidney injury development
chymase hydrates Ang
Renal chymase-dependent pathway
ACE
aristolochic acid I-induced
CI
Ang II
nephropathy Angiotensin-converting enzyme
C 57BL mice
AAI-treated mice
role
AAI-induced nephropathic mice
Renal RAS-related enzyme activities
2019-01-11 18:36:51
Figure
https://plos.figshare.com/articles/figure/The_level_of_inflammatory_signaling_pathways_in_the_kidneys_of_mice_with_AAI-induced_acute_nephropathy_/7579643
<p>The expression of p-MEK (<b>A</b>), p-STAT3 (<b>B</b>) and p-Smad3 (<b>C</b>) in kidney tissues was determined using Western blotting and representative results of Western blotting are shown (<b>D</b>). All values are expressed as the means ± SD from each group (n = 9). * and ** indicate <i>p</i> < 0.05 and <i>p</i> < 0.01 compared to the Control group, respectively; †† indicates <i>p</i> < 0.01 compared to the AAI group.</p>